Study for the Medical FIDSAP v2 Test. Sharpen your skills with flashcards and multiple-choice questions, complete with hints and explanations. Prepare effectively for your upcoming test!

Multiple Choice

A 59-year-old male with a newly replaced central venous catheter develops chest pain, shortness of breath, cyanosis, tachycardia, and hypotension. What should you suspect?

When a patient with a central venous catheter develops sudden chest pain, shortness of breath, cyanosis, tachycardia, and hypotension, think of an air embolism. Air can enter the venous system if the catheter is open to air or during insertion or removal, and it travels to the right side of the heart and then into the pulmonary circulation. This obstructs blood flow and causes acute hypoxemia and hemodynamic instability, which fits the rapid, systemic symptoms you’re seeing here. What to do next is aimed at removing the air and supporting circulation: place the patient in the left lateral decubitus position with the head down (Durant maneuver) to trap air in the right atrium and prevent it from entering the pulmonary arteries, give 100% oxygen to help wash out the nitrogen from the embolus and improve oxygenation, and stop further air entry by securing or removing the catheter if needed. If accessible, attempt to aspirate air through the central line. Provide IV fluids and vasopressors as needed to maintain perfusion. Other options don’t fit as well because they typically cause local signs or catheter malfunction rather than this abrupt, life-threatening systemic presentation. Phlebitis presents with warmth and redness along the vein; catheter occlusion causes reduced infusion or drainage but not this acute shock picture; catheter migration might cause arrhythmias or loss of catheter function, but not the immediate cardiopulmonary collapse seen here.

When a patient with a central venous catheter develops sudden chest pain, shortness of breath, cyanosis, tachycardia, and hypotension, think of an air embolism. Air can enter the venous system if the catheter is open to air or during insertion or removal, and it travels to the right side of the heart and then into the pulmonary circulation. This obstructs blood flow and causes acute hypoxemia and hemodynamic instability, which fits the rapid, systemic symptoms you’re seeing here.

What to do next is aimed at removing the air and supporting circulation: place the patient in the left lateral decubitus position with the head down (Durant maneuver) to trap air in the right atrium and prevent it from entering the pulmonary arteries, give 100% oxygen to help wash out the nitrogen from the embolus and improve oxygenation, and stop further air entry by securing or removing the catheter if needed. If accessible, attempt to aspirate air through the central line. Provide IV fluids and vasopressors as needed to maintain perfusion.

Other options don’t fit as well because they typically cause local signs or catheter malfunction rather than this abrupt, life-threatening systemic presentation. Phlebitis presents with warmth and redness along the vein; catheter occlusion causes reduced infusion or drainage but not this acute shock picture; catheter migration might cause arrhythmias or loss of catheter function, but not the immediate cardiopulmonary collapse seen here.